Article
Friday Pearls
Supplement Label Claims
May 24, 2012
In spite of peer-reviewed evidence suggesting over 90 percent of the US population suffers from nutritional deficiencies that contribute to degenerative diseases, current FDA law prevents micronutrient formulators from making science-based label, brochure, or web site claims suggesting that dietary supplements can prevent, treat or cure any disease. Last year, Americans spent around $200 billion on FDA approved prescription drugs, which are allowed to claim that they cure or safely managed disease processes, despite mounting clinical evidence to the contrary in many cases.
Overwhelming amounts of scientific evidence now support the fact that micronutrient deficiencies impact genomic stability, including exposure to carcinogens, DNA repair, DNA synthesis and apoptosis. Because diseases of development, degenerative diseases, and aging itself are partly controlled by damage to DNA, it seems logical that the FDA would direct new science to focus far more attention on redefining optimal requirements of inexpensive key vitamins, minerals and antioxidants that prevent damage to both nuclear and mitochrondrial DNA.
The list below contains a few of the most elementary and medically accepted micronutrient claims that our esteemed FDA could not deem entirely legal under current law, even though abundant published peer-reviewed science strongly suggests otherwise:
1) Vitamin C prevents and cures scurvy.
2) Vitamin D prevents and cures rickets
3) Vitamin B1 prevents and cures beri beri
4) Vitamin B3 prevents and cures pellagra
5) Vitamin B12 prevents and cures pernicious anemia
6) Folate prevents neural tube defects in the newborn.
Unfortunately, there are still no micronutrient or lifestyle choices routinely recommended by most eye doctors for cataracts, glaucoma, diabetic retinopathy, or macular degeneration prevention, despite large amounts of supporting science.
PEARL
References
Micronutrients and genomic stability: a new paradigm for recommended dietary allowances (RDAs). Fenech, M. Food Chem Toxicol, 2002 Aug;40(8):1113-7 [ abstract]